Nursing Ethics in Mental Health Capacity and consent

Nursing Ethics in Mental Health Capacity and consent

 

 

Nursing Ethics in Mental Health

Capacity and consent

• Refusal of treatment

• Voluntary vs. involuntary admissions

• Myths and stigma

Learning Activity: Case analysis

The Scott Starson case:

https://www.jemh.ca/issues/v3n2/documents/JEMH_v3n02_article_The_Delicate_Dance_in_Canadian_Mental_Health_Policy.pdf

Mental health and Community Treatment Order information. Mental Health and Law Toolkit, 2012, Ontario Hospital Association, pages 1-6

NOTE: SPEAK AGAINST THE MOTION BY REPRESENTING THE ONTARIO CONSENT AND CAPACITY BOARD. (explain the role of Board also)

Gives reasons why you are against the motion.

Motion: We support the Supreme Court decision in the Starson Case.

(Mr. Starson has capacity to make his own treatment decisions)

PLEASE USE Canadian Nurses Association (2008). CODE OF ETHICS AND Canadian Nurses of Ontario (2009) AND OTHER BIOETHICS JOURNALS. ADD and incorporate ETHICAL PRINCIPLES which correlate to the paper, such as paternalism /materialism beneficence, non-maleficence etc. AND THEORIES such as rule utilitarianism into the research. Thank you.

Identify antecedents and consequences

Identify antecedents and consequences

For steps in the Process –see Chapter 3-McEwen & Wills (2011) and Chapter 10 Walker & Avant Handour Introduction (Purpose of the paper) Dictionaries Definitions Review of Literature Nursing implications Defining Attributes Definition of the Concept Identify (write) a model case 6. Identify (write) a borderline, related, contrary, invented and ileegitimate case 7. Identify antecedents and consequences 8. Define empirical referents

Read a qualitative nursing study.

Read a qualitative nursing study.

question 1: Read a qualitative nursing study. If a different investigator had gone into the field to study the same problem, how likely is it that the conclusions would have been the same? How transferable are the research's findings?

Question 2: You have been asked to be a peer reviewer for a team of nurse researchers who are conducting a phenomenological study of the experiences of physical abuse during pregnancy. What specific questions would you ask the team during debriefing and what documents would you want the researchers to share?

The Deep Vein Thrombosis Health And Social Care Essay

What is Deep Vein Thrombosis or well known as DVT. Did you ever heard about blood clot? A condition which a blood clot thrombus forms in a vein is known as venous thrombosis. Blood flow through the vein can be limited by the blood clot, resulting in swelling and pain. Most commonly occurs in the deep vein in the legs, thigh or pelvis but it can still happen elsewhere in the body (Pai and Douketis, 2012). The larger veins that go through the muscles of the calf and thigh are deep leg veins. They are not the veins that we can see just below our skins, neither are the same as varicose vein. Deep Vein Thrombosis is most common in adults over age 60 but it can happen at any age as well. DVT usually can cause embolism when a part or all of the blood clot in the vein breaks off from the site where it is formed and travel along the venous system. DVT can lead to long lasting problem. It can damage the vein and cause the leg to ace, swell, change color and leg sores after years.

What cause deep vein clots to form? Blood clot can form in veins when you are inactive. For instant, clots can form if you are paralyzed or sit while on a long journey. Surgery, injury and cancer also can damage your blood vessel and lead to blood clot. If DVT remain in the legs it can cause a few complications including phlebitis and leg ulcer also can lead to pulmonary embolism. Phlebitis is a condition which blood clots with inflammation in superficial vein was rarely cause serious problem but if blood clot in deep veins happen require instant attention because it can lead to embolism.

Deep Vein Thrombosis can cause the blood flow in the vein is partially or completely blocked by the blood clot. The common site for DVT is in calf vein and a thigh vein is less commonly affected while DVT is rarely happen in other deep veins. There are few alternative names for DVT such as thromboembolism, post-phlebitic syndrome or post-thrombotic syndrome. A pulmonary embolism is a life-threatening complication and long-distance flights may contribute to the risk of DVT or also known as economy-class syndrome. Coronary heart disease, being overweight or obese, cigarette smoking, pregnancy, family history of DVT or recent surgery or injury also can lead for DVT to happen. A DVT is often just a one-of event after a major operation has been done. However, some people who develop a DVT have an ongoing risk of a further DVT. If have a blood clotting problem or continued immobility, then everybody are advised to seek for a medical care or take anticoagulation such as heparin injection (after which they are prescribed warfarin) to avoid further complication.

LITERATURE REVIEW

2.1 Histopathology

Histopathology refers to the microscopic examination of tissue in order to study the manifestations of disease. Examination of a biopsy or surgical specimen by pathologist, after the specimen has been processed and histological sections have been places onto glass slides also can well describe about the histopathology.

Regarding with DVT, its histopathology is quite complex to understand. Differential diagnostic considerations prior to thrombolytic treatment and surgery should include tumours. Definitive diagnosis can be achieved by a biopsy but CT and MRI also bring quite a role in diagnosing DVT.

However, CT and MRI just such a waste when the disease is at an advanced stage because any of these examinations should be done in the early stage of disease. Based on Phlebol (2006), soleal vein was the most frequent site of DVT. At first, primary thrombi would be formed at soleal veins, then its will propagate to proximal veins. The proximal veins would be occluded by fresh thrombi, thereafter secondary thrombi were made at non-drainage calf veins. Paterson and McLachlin found that most venous thrombi consisted of two regions. One of it is composed predominantly of fibrin and trapped erythrocyte while the other one are composed mostly by aggregated platelets. The fibrin-rich regions that attached the thrombi to the vessel wall, while the platelet-rich regions localized further from the site of attachment. These show that activation of coagulation system come before platelet activation and collection during the formation of venous thrombi (Lopez et al, n.d). Based on that information, we know that the use of anti-platelets drug in venous thrombosis is very limited. Histopathology evidence in DVT shows that coagulation occurs on or nearer to the endothelial surface. When coagulation starts on the endothelial surface, platelets may be regrouped to the fibrin clot rich in thrombin through adhesive interactions and it will result to further thrombus growth.

Based on everything that stated above, we can say that the platelet collection localize to regions of the clot that are far away from its site of attachment and anti-platelet drugs such as aspirin has prove that it can reduce the risk of DVT in our precious life.

2.2 Causes and Risk Factors

Deep Vein Thrombosis occurs when a blood clots forms in a deep vein in our body. DVT always happen in the legs but it can still happen in your arms, chest, or other areas of your body. The blood clot can block our circulation or lodge in a blood vessel in our lungs, heart, or other part of our body and can cause severe organ damage and can lead to death. This topic will reveal about every causes and risk factors that can lead to DVT.

There are many causes and risk of DVT. Some of that are:

A person will have DVT when a vein’s inner lining is damaged. There are many factors that can lead to this injury. For instant it can be cause by physical, chemical, or biological factors. Besides, surgery, serious injuries, inflammation and immune responses also can be the causes to it.

DVT can also happen when the blood flow is sluggish or slow. Immobility or lack of motion can cause sluggish or slow blood flow. This condition always occurs after the surgery, bed rest for a long period and having a long journey that take a long time.

A condition which blood is thicker or more likely tend to clot than normal (thrombophilia) also can result in DVT. This is due to inherited condition such as V Leiden factor that increase the risk of blood clotting. Apart from that, hormone therapy or birth control pills also can increase the risk of blood clot.

The contraceptive pill and hormone replacement therapy (HRT) has small increased risk of DVT since the oestrogen in it can cause the blood to clot slightly more easily.

People with cancer or heart failure can also increase the risk for DVT. Usually, investigation looking for the cause of DVT may show cancer to be the underlying cause.

Older people over the age 60 years also likely to have DVT particularly if they have poor mobility or having a serious illness that can stop them to do a lot of action.

Pregnancy also increased the risk for DVT to happen. Normally, within six month after they give birth or while they are pregnant.

Dehydration will increase the chances for DVT because the blood becomes more sticky an liable to clot.

As a male, precaution should be taken because men tend to develop a DVT more often than women.

Being an obese person also can lead to DVT.

There are many causes and risk of DVT that we are unaware of it for the certain time.DVT can happen anywhere in our body part and also can attack everybody in different ages but older people are more vulnerable to it. The most risky patient to have DVT is after having a surgery because the blood can easily clot if it not cared in a good ways. Lack active persons also are in a high risk of DVT since it will cause the blood to flow slowly and easily to clot. DVT also can be inherited and wrong pill intake also can result in DVT. In easy word, there are many causes and risks that can lead to DVT and every citizen around this world should take every safety precaution to avoid DVT.

2.3 Incidence and Comparison

There are many people around this world that have experience DVT. About 2 million Americans have experienced DVT each year without they are realizing it. Based on Convenient option for DVT (2012), the exact incidence of DVT is still unknown in Malaysia but there is growing evidence that DVT is not uncommon in Asians. Based on autopsy studies, hospital audits of admission to major hospitals and also subclinical DVT in high risk situations such as after major joint surgeries show that there is increment of the incidence. According to Prof Hatem Salem, Head of Department, Australian Centre for Blood Diseases, there is grave misconception that DVT is rare in Asians because he finds out that Asians too are at risk of DVT (Convenient option for DVT, 2012). Commonly, DVT happen after post-surgeries and a few reports have appeared with high incidence of DVT in orthopedic patients comparable to Western study. Dhillon, Askander and Doraisamy (1996) suggest that the present practice of withholding routine prophylaxis against thromboembolism in Asian patients undergoing high-risk orthopaedic procedure should be reconsidered.

In Western countries, DVT occurs in 45% to 84% of patients after hip and knee surgery in the absence of prophylaxis (Stulberg et al, 1984) but there is a firm belief that the complications is quite rare in Asian patients. Lack of awareness in Asia of a condition that become one of the main killer factors in West is due to the faith that thromboembolic disease is rare in Asia. Since DVT always have been linked with post-operative so every patient that have undergoes surgery should take a good care of their health to avoid DVT. However, there are few opinions that stated DVT is rare in Asians and the first report was made by Tinckler in 1964 stated that there is rarity of post-operative DVT and pulmonary embolism in Asians (Tun et al, 2004). A study that has been made in a few Asians country like Malaysia, Hong Kong and Japan also show that there is low incidence post-operative DVT has happened around this country.

A few incidences in Asians can be taken to make a comparison with the Western to show differences in frequency of DVT in this world. In developed countries of the Western area show that DVT and consequent pulmonary embolism is still becomes the number one threat to post-surgery while in Asian specifically in Malaysia show that there is still low incidence of DVT after the operation done.

A study has been made in United Kingdom to represent Western hemisphere and Malaysia as Asian’s representator. In UK, Sandler and Martin found that 9% of patients admitted to a general hospital died and 10% of these deaths were due to pulmonary embolism that originated from DVT of lower limb. Based on a study made in Hospital Universiti Sains Malaysia, Malaysia on 45 patients, only one positive DVT confirm among 45 patients that have been observed. There is only 2.2% and this good result show incidence of DVT among patient in Asia is still low (Tun et al, 2004).

In a nut shell, the incidence of postoperative DVT in Asian patients is not low as is commonly believed and also it is not high like we know. Larger studies are needed to settle this controversy and find out all the true fact regarding this matter. Based on study that has been made above, routine practice of withholding prophylaxis in Asian patients undergoing high-risk orthopaedic procedure should be reconsidered. We can conclude that DVT is still low in Asians but we should be aware of DVT in the future because it is too risky to take this matter as small things.

2.4 Mortality and Morbidity

If DVT is left untreated, there are many bad effects can happen and some of that can result in mortality and morbidity. There is short-terms morbidity in DVT such as cardiopulmonary consequences that may delay weaning from mechanical ventilation and there is also long-term morbidity like patient-centered consequences such as chronic venous insufficiency. Based on Vascular Medicine (1998), short-term mortality for DVT patient is reported to range between 7% and 15% only while long-term mortality has record a great number of deaths for patient with DVT. In a Dutch study of 355 patients, 90 died during follow up. Patients with a DVT are at risk for morbidity and mortality since a fragment of the thrombus can embolize to the lungs. Anthony and Bon (2004), suggested that about one half of patients with an untreated proximal DVT will develop a pulmonary embolism within 3 months. In the past, contrast venography has been used to rule out DVT. Nevertheless, due to some problem such as expenditure of manpower and time, space and equipment and most importantly is it also associated with morbidity, it was been terminated and been replaced with other machine that can overcome this problem. There are many indications of short-term mortality of patients with DVT such as cancer, pulmonary embolism and major bleeding. There are also many caused that can lead to long-term mortality such as malignancy, pulmonary embolism, acute myocardial infarction, ischemic stroke and anticoagulant related to hemorrhage.

2.5 Pathophysiology

2.6 Signs and Symptoms

There are few symptoms to recognize DVT but often DVT occurs without any symptoms. The symptoms of DVT are related to obstruction of blood returning to the heart and causing a pooling of blood in the leg. Patient with DVT will undergo swelling of the affected leg and the leg may feel warm and look reddish. Apart from that, patient calf or thigh may ache or feel tender if it is been touch or squeeze or when stand or move. There are no symptoms appear if the blood clot is small and for some cases, Pulmonary Embolism is the first sign that confirm for DVT. Basically, it can be hard to detect DVT since some of the symptoms are same with other health problems. Sign and symptoms alone are not enough to determine the DVT but when risk factor is take under considerable, then it can help to determine likelihood of DVT.

Some of the common sign and symptoms of DVT:

Pain

Swelling(edema)

Tenderness

Redness or skin color changes

Skin warmth

Discoloration

Distention of veins surface

Discomfort when the foot is pulled upward

Leg fatigue

Signs and symptoms occur vary depending on the severity of the condition and not all of these symptoms have to occur with deep vein thrombosis.

2.6.1 Conditions That May Cause Similar Symptoms

Patient is advised not to make any early assumption in having Deep Vein Thrombosis if they are undergo the symptom that stated above since there are a number of different conditions that can cause the same sign and symptoms like DVT.

Some of the conditions are:

Muscles aches and tears

Superficial thrombophlebitis (blood clot that forms in an inflamed part of a vein near the surface of the body)

Varicose veins (blood vessels that are abnormally swollen and twisted

Blood clots in arteries

Arthritis (inflammation of the join)

Cellulitis (infection in tissue under the skin)

Bone fracture

Lymphedema (swelling in the hands and feet caused by excess fluid retention)

Since DVT symptoms are quite same like other health problem, patient need to undergo specific procedure and special test to confirm the diagnosis or rule out the other problem.

IMAGING MODALITIES

3.1 First Line Evaluation

3.2 Second Line Evaluation

IMAGES FEATURE OF PATHOLOGY

TREATMENT AND PREVENTION

PROGNOSIS

CONCLUSION

Week 4 assignment: source evaluation

Instructions

Goal: The goal of this assignment is to research your topic, evaluate selected sources, and organize your sources.

Description: During the Week 2 Assignment, Project Plan, you chose a topic and created your project plan. In the Week 3 Lesson, you read about location and access and in Week 4, you learned about organizing your digital information and storing that information responsibly. Now it is time to take the work you did in the Week 2 Assignment, Project Plan, and locate and evaluate sources  (CO2 & 5) that that will lead to your Week Week 7 Assignment, Field of Study Project.

Please follow these instructions: – Download the Source Organization Worksheet Template (in Word).- Complete Parts I and II in the Template. – Find a minimum of three (3) sources. One (1) source must be an academic source and from the APUS Trefrey Library and the other sources must be credible and appropriate for college research.- Please include robust source summaries that explain the contents of the source and the relevance of the source to your topic.

Consider the following: – Once you complete the Source Organization Worksheet Template, please submit the completed Worksheet.- See the attached rubric for grading guidelines.

Essay on Leukemia

Leukemia is a common cancer of the bone marrow. In the United States it occurs mostly in the young and elderly and is associated with down syndrome. This research will go over the back-ground information on leukemia, types of leukemia, demographic, genetic links, and treatment for each types of leukemia.

There are 3 types of blood cells: erythrocytes, leukocytes, and thrombocytes. The body forms these cells in the process known as hematopoiesis.  Hematopoiesis occurs in the bone marrow, although in fetus it occurs in the liver and spleen (Huether, 497). Leukemia is a cancer which alters the bone marrow to produce abnormal leukocytes known as the leukemic cells (Huether, 564). Leukemia is characterized by the rapid increase of leukemic cells which occupies the bone marrow and lead to less production of normal functioning blood cells (Huether, 526). Leukemia can become malignant fast because leukemic cells can spread quickly to another part of body through the blood vessels.

To understand the different types of leukemia, we need to understand the differentiation of leukocytes. When bone marrow undergoes hematopoiesis, it produces hematopoietic stem cells which can differentiate into erythrocytes, leukocytes, or thrombocytes, depending on the path it takes. To become leukocytes, hematopoietic stem cells can either differentiate into myeloid stem cells or lymphoid progenitor cells. If it differentiates into myeloid stem cells, it can further differentiate into granulocytes and phagocytes. If it differentiates into lymphoid progenitor cells it will than become lymphocytes, which possesses more specific immune functions (Huether, 494).  Often, leukemia occurs due to abnormal or translocation of chromosomes. To classify leukemia, we look at the cells of origin which is myeloid stem cells or lymphoid progenitor cells. We also look at the rate of progression which can be acute or chronic. Usually when dealing with leukemia that has a sudden onset which involves undifferentiated cells, it is considered acute. Chronic leukemia usually deals with more differentiated cells and will progress more slowly. There are four types of leukemia in total: acute lymphocytic leukemia (ALL), acute myelogenous leukemia (AML), chronic lymphocytic leukemia (CLL), and chronic myelogenous leukemia (CML) (Huether, 526).

ALL is the most common leukemia seen in children. According to American Cancer Society, it is more prevalent in woman and the risks increase for children under five and older adults over 50. Death from leukemia increase greatly in elderly because they’re not able to withstand the damage done by aggressive treatment, whereas children are able to cope with it much better. ALL is uncommon type of leukemia and it is more common in Whites and Hispanic and less in Blacks (American Cancer Society, 2018). AML is an acute form of leukemia that is more likely be seen in an adult. Unlike ALL, male has more prevalence in being diagnosed with AML but it is more commonly seen in Whites than Blacks as with ALL. Interestingly, risks for developing this type of leukemia greatly increase as household income increases (Goyal. G, 2015). In general, acute leukemia has high prevalence in Whites population and less common in Eastern Europe, Asia, and Central America. However, this doesn’t include Japan due to atomic bombing that occurred during WWII (Huether, 528). Chronic leukemia occurs mainly in adult especially after the age of 40 and rarely in children (Huether, 530). CLL will more likely to develop in men than in women (American Cancer Society, 2018) and increase prevalence in an individual older than 70 (Huether, 530). CML is also seen more in men than in women and also increase prevalence as we age (American Cancer Society, 2018).

In ALL, there is an abnormal increase in the production of altered lymphoid progenitor cells due to alteration caused in bone marrows. Signs and symptoms become apparent suddenly. Patient may experience fatigue due to anemia. Anemia is present because leukemic cells accumulates in the bone marrow and lead to decrease production of healthy erythrocytes. Hemorrhage also occur due to thrombocytopenia where there is decrease in production of thrombocytes. Fever is also common due to increase in susceptibility to infection. This is because normal functioning leukocytes are reduced which prevent them from fighting pathogens that are harmful to our bodies. Other symptoms may be experienced such as headache, blurred vision, hepatosplenomegaly, and vomiting (Huether, 529). The cause of abnormalities seen in ALL is due to genetic abnormalities such as aneuploidy (Woo, 2014). Aneuploidy is when cells does not contain the normal number of chromosomes which is 46 or 23 pairs. There are different types of aneuploidy such as trisomy and monosomy. In trisomy, there are three pairs of chromosomes so in total there are 69 chromosomes within a cell. Monosomy on the other hand, only contain 23 chromosomes which are not paired (Huether, 42-43). According to NCBI, hyperdiploidy is the most common structural chromosomal arrangement that leads to ALL. Hyperdiploidy occur when there are more than one chromosomes that contain more than one pair. It occurs mainly on chromosomes 4, 6, 10, 14, 17, 18, 21, and X. High hyperdiploidy is associated with alleles PRDM9 which will control the recombination of chromosomes. Hypodiploidy can also lead to ALL although it only affects about 5% of ALL patient (WOO, 2014) Genetic abnormalities also can be due to different arrangements of chromosomes such as translocation where there is change of genetic materials between two different chromosomes (Huether, 48). For example, one translocation that can occur is between chromosome 12 and 21 which occurs in 25% of ALL patients. The section p13 of chromosome 12 which encodes for ETV6 and section q22 of chromosome 21 that encodes for RUNX1 exchange their materials and lead to production of altered lymphoid progenitor cells. Transcription factors ETV6 and RUNX1 are both needed to maintain normal hematopoiesis, however, fusing those two together will disrupt the normal function of these proteins. Deletion or rearrangement can also occur in chromosomes such as chromosome 9p13 which is transcription factors for PAX5 which is also needed for normal hematopoiesis. 32% of patients with ALL are associated with PAX5 mutation (Woo, 2014). Epigenetic factors may also be associated with development of ALL. Epigenetics are turning on and off the genes without actually causing any alteration to the DNA (Huether, 2017). For example, microRNAs regulate the differentiation of hematopoietic cells and its proliferation which may be promoting the development of leukemia. Thankfully there are 90% survival rate for children who suffer from ALL. However, it is very likely to relapse and the survival rate will decrease to 30% the second time. This is most likely due to increase in damaged and mutated cells due to progression and treatment of disease (Woo, 2014).

In AML, there are significant increase in production of myeloid stem cells, which also accumulate in bone marrow and greatly reduce the number of production of other healthy blood cells. Alteration in transcription factor CEBPA is the usual cause of AML and is genetically passed down in autosomal dominant form. CEBPA transcribe into CCAAT enhancer-binding protein alpha which is needed for differentiation within bone marrow and thought to be tumor suppressor genes as well. Therefor alteration in CEBPA is associated with uncontrollable proliferation of leukemic cells. Signs and symptoms are similar to those of ALL. Infection may occur more easily since alteration of CEBPA leads to leukopenia and lose the army to fight off infection. Survival rate for AML is less promising. If it is due to alteration of CEBPA, the survival rate is about 60%. If it’s due to other causes it drops dramatically to about 37.5% (NIH, 2015).

The most common form of adult leukemia is CLL. The most common cause of CLL is due to deletion of 13q14 chromosome which is also associated with tumor suppressor genes. Surprisingly there are no or only some other chromosomal changes for CLL which indicates that the deletion of chromosome 13q14 is significant in developing CLL. A research found that large chunks of 13q14 was deleted and with further investigation was able to determine that this deletion occur specifically within LEU2 genes of region 29-kb between 2-5 exons. Furthermore, we found that miRNA15 and 16, which are needed for normal lymphocyte production, reside within this deleted region and concluded that this must be connected to development of CLL. As it was hypothesized, studies have found that at least 68% of patients suffering from CLL do in fact has reduction of miRNA15 and 16. It is also found in many other diseases and therefore, we can say that deletion of miRNA15 and 16 have pathogenetic consequences (Croce, 2002).

CML is another chronic, slow developing leukemia usually seen in an older adult. As seen in all other types of leukemia, similar signs and symptoms can be seen. However, in chronic leukemia, as with CLL, when patient is diagnosed with leukemia, they may not have any apparent symptoms. There are three phases to CML. First is the chronic phase. In this phase, there are increase in proliferation of matured leukemic cells and there is significant decrease in the number of myeloid stem cells. This phase last for long periods of time and many patients won’t express any symptoms. The second phase is accelerated phase where myeloid stem cells number slightly increase and symptoms starts to be apparent. This phase is shorter, lasting up to 6 months or may not occur in some patients. The last phase is known as blast crisis where patients experience severe signs and symptoms and is life threatening. The cause of CML is not due to genetic factor and is due to translocation chromosome 9 where it encodes for ABL1 gene and chromosome 22 where it encodes for BCR gene. Individually, BCR gene is known to code for protein that is responsible for cell signaling. ABL1 gene is important for proliferation, differentiation, migration, and apoptosis. When these two genes fuse together, it forms an abnormal gene BCR-ABL1 which is known as Philadelphia chromosome and will promote proliferation and diminish apoptotic function. The proliferation of leukemic cells are so severe in CML that normal blood cells almost diminishes (NIH, 2016).

Treatment for leukemia is mainly done by prolong, aggressive or non-aggressive chemotherapy (American Cancer Society, 2018). Chemotherapeutic agents are used in chemotherapy where each agent will attack specific weak spot and shrink the cancer cells. Therefore, using multiple agents for treatment shows best result, however, because it is also harmful to healthy cells and there are many side effects, not all patients can undergo such harsh treatment (Huether, 260). Chemotherapy is done in three stages. Complete remission is the main goal for the first stage known as induction. During this process patients may stay or spend most of their time in the hospital to prevent developing infection. This process can be quite intense and requires patient to have the therapy done frequently. The second stage is known as consolidation/intensification which is the next treatment that patients go through after they are in remission. Due to high risk of leukemia relapse, this stage continues using high dosed chemotherapeutic agent. Stem cell transplant may also be used to prevent relapse of leukemia. The last, longest stage is known as maintenance which takes in average of two years. According to American Cancer Society, remission of leukemia in adult is very promising although there are high prevalence or relapse seen. If there are no improvement seen by chemotherapy, antibody administration to fight infection may be used to subset signs and symptoms. If there is no available way to treat leukemia, palliative care is available for patients which is to control the symptoms and manage pain (American Cancer Society, 2018).

There are few new preventions and treatments that healthcare providers are trying to invent to improve the management and survival rate of leukemia. For example, there are researchers who found a way to identify risks up to five years before the development of AML. This is surprising because acute leukemia develops suddenly but give the healthcare provider hope to detect this disease early enough to decrease the probability of developing this disease. Researchers have used AML patent’s stored DNA and compared it to individuals who are not diagnosed with AML. We have found that there is translocation of chromosome which encode for ARCH gene and alteration in this gene increase with age. Fusing of genes create abnormal gene known as ARCH-PD which was found in 73.4% of individuals who are at high risk of developing AML (Schieszer, 2018).

Another example is the third generation of Anti-CD20 monoclonal antibodies which are specifically developed to treat CLL which is undergoing clinical trials. Targeting antiapoptotic proteins, this antibody allows normal functioning apoptosis in bone marrow and prevent proliferation of leukemic cells (Robak, 2014).

As we can see from this research, leukemia is a serious complication that is commonly seen in United States. Patients can achieve complete remission in many cases of leukemia, however, it is also common to relapse with reduced survival rate. Treatments can be very harmful to our body and although many patients benefit from it, palliative care may be the only option left for some patients. There are many treatments that is being developed for clinical trials and we are hopeful to see improvement in increasing survival rate for leukemia.


References

Comparison of Public Health and Clinical Research Ethics


How Does Public Health Research Ethics Differ From Clinical Research Ethics?


  • Zoheb Rafique


INTRODUCTION:

The field of biomedical ethics arose in late 1960s in the response to some emerging ethical dilemmas and issues of that era. This field for many years focused on dilemmas generated by the high technology medicine, rather than on the issues of the population health and the ethical problems of public health programs. The discipline Bioethics received the initial stimulus from abuses and issues of human subject’s research, also the emergence of patient’s rights movement, and the drama of high technology medicine. The Research involving human subjects has often been a central ethical issue and problem for the biomedicine for at least hundred (100) years now, and particularly since the World War 2. Just as the public health is broad in the scope, the range of the ethical issues in this field is uncommonly wide, and encompassing the ethics in the public health as well as ethics of public health (1). In past twenty (20) years, the research base of the clinical ethics has gained tremendous strength appreciably. However the main research opportunities didn’t come under broad heading of the clinical ethics, but instead through the specific programs such as human genome project and end of life movement (2). In this paper, I will discuss the differences between public health research ethics and clinical research ethics.


DISCUSSION:

The Public health research ethics include ethics regarding the community participation in the research, while the clinical research ethics include the ethics regarding the clinical patient research. This is major difference between these two scenarios, now I will discuss it in detail. Most of the research has focused on the clinical and the experimental medicine (efficacy, safety and the mechanism of action, and also regulatory issues to general neglect of the public health dimensions. The Public health ethics, which is defined as identification, analysis, and the resolution of the ethical problems occurring in the public health practice and also research, and it, has different domains than those of the medical ethics. The ethical concerns in the public health often relate to dual obligations of the public health professionals to apply and acquire the scientific knowledge that aimed at restoring and also protecting public’s health while respecting the individual autonomy. Ethics in the public health involves interplay between safeguarding welfare of individual, as in medicine, and the public health goal of protecting public welfare. Some other ethical concerns in the public health relate to need to ensure just distribution of the public health resources. The Public health ethics has broad scope that includes the ethical and the social issues arising in the health promotion and the disease prevention, the epidemiological research, and also public health practice. The main professional roles of the epidemiology are design and the conduct of the scientific research and public health application of the scientific knowledge. This includes reporting the research results and also maintaining and promoting the health in communities. Also in carrying out the professional roles, the epidemiologists often encounter many ethical issues and some concerns that require the careful consideration. Many of those issues have been highlighted and addressed in literature on ethics in the epidemiology and the public health including the ethics guidelines. The Ethical and the professional norms in the epidemiology have also been clarified in the ethics guidelines for the epidemiologists and often the public health professionals. The Ethics guidelines such as those developed for Industrial Epidemiology Forum, International society for the Environmental Epidemiology, and American college of the Epidemiology also provide useful accounts of the epidemiologists’ obligations to the research participants, employs, society, and colleagues (3). The Epidemiologic studies can provide the descriptive data that can lead scientists later to develop some intervention that can result in the reduction in morbidity and mortality; the health education program can be one of the multiple interventions that together reduce the risks and also ill health. The argument here, however, is that the public health programs, studies, or interventions, must be designed with the awareness of relationship between that program and ultimate reduction in the morbidity and mortality. The Public health programs may result in high employment, as well as some less tangible benefits such as coalition building and strengthening of the communities. Today, the public health practitioner use some tools in addition to the epidemiology to register their work, still aiming primarily on community wide, also typically prospective methods and approaches to improve health. In addition, the practitioners investigate the outbreaks, provide health education, conduct contact tracing, and also other preventive interventions, and organize research related to the public health (4). The Public health agencies require the identifiable health information for conducting different public health activities. The increasing number of the functions, including the public health Surveillance, and outbreak and incident investigations and program implementation, and some direct health services, such as the clinical public health activities and services and the research, maintenance, and the storage of the personal health information. The Successful execution of all these functions depends on the data quality and the accessibility. Heightened security is very necessary and paramount to maintain the public confidence; also good health care and it depends on the patients providing the accurate and sensitive information to their care providers in a very timely manner. Placing restrictions on the data acquisition, use, and the disclosure also poses some risks, particularly if those restrictions impede acquisition of the key surveillance data, which would otherwise be used to prevent the disease, investigate the causation, and enable the interventions to protect the exposed population. Additionally, electronic data could potentially permit real time public health Surveillance and also can facilitate the faster emergency response (5). Advances in the science, technology and the biomedical research have pushed the boundaries of Belmont principles and stimulating the need for the communities to be involved in informed consent process. Changes in the Food and the Drug Administration regulations allow the waivers of the informed consent in life threatening emergencies. The rights of the unconscious participants are assumed to be accorded degree of the protection through mechanism of the “Community Consultation” which requires the prior consultation by the investigators and the institutional review board with the community representatives and public disclosure to affected community both before and after that research (6). Now I will discuss the clinical research ethics, and we will see how it is different from public health research ethics. Taking into account the sound and the increasing emphasis of recent years that the experimentation in man must precede the general application of the new procedures in the therapy, and also there is reason to fear that these requirements and the resources might be greater than supply of the responsible investigators. Medical schools and the university hospitals are increasingly dominated by the investigators. Every young man knows that he will never be promoted to some tenure post, and to a professorship in a major medical school, unless he has proved himself as an investigator. If the ready availability of the money for conducting the research is added to this fact, one can see how great the pressures are on the ambitious young physicians (7). A taxonomy was developed for the clinical ethics research, based on the method rather than the clinical area. This divided research in different terms of whether it used theoretical or any empirical methods. First, we will see the theoretical methods of the clinical ethics research. Philosophy (e.g., How should the decisions on setting the priorities be made legitimate and also fair?). Law (e.g., what practices in setting the priorities in regional health authority might constitute discrimination?). Policy (e.g., what policy should the governments follow in funding the new technologies in medicine?). Now let’s see the empirical methods of clinical ethics research. Social Sciences (e.g., how do the regional health authorities in the developing countries make the decisions on setting the priorities?). Decision analysis (e.g., How do you trade-off considerations of equity and efficiency in the decisions on setting priorities?). Clinical epidemiology (e.g., what are the criteria used to allocate the liver transplant?). Health services research (e.g., how does the delivery of the cardiac surgery vary by patient gender and ethnicity?). Within empirical research (both in ethics and more generally), there is some growing recognition that the quantitative methods alone are not adequate. Since many of the phenomena examined by the ethics researchers are deeply entwined into fabric of professions, organizations, and the human lives, qualitative methods have begun to play an important role. For example, one investigator performed the observational research on how physicians discuss do-not-resuscitate orders and also advance care planning. The role of the qualitative methods is both increasing and broadening to include not only the content analysis but also grounded theory, the ethnography, and the case study designs. When we review the field of the clinical ethics a decade from now, we hope that the focus will have shifted from the ethics courses, committees and the consultants to an understanding on the part of most physicians and medical students that ethics is an inherent and inseparable part of the good clinical medicine. We hope that clinical ethics will have achieved its rightful place at the interstices of relations between the patients who are sick and physicians who profess to be able to heal and comfort them. Clinical ethics has made progress towards this vision in the past some years. The challenge remains for the research into ethical issues to become a mainstream concern for the funding agencies around the World.


CONCLUSION:

In conclusion, it is stated that although public health research and clinical research are different from each other, but ethical dilemmas are faced by both and also they are same in many situations. While considering the public health research ethics, the researcher must show respect for community’s culture, also take community input on the protocol development, and ensure that research is useful to community, and should respect the community’s knowledge and the experience, and ensure that the informed consent is correctly taken before starting any of the research (8). While considering the clinical research ethics, two components are most important, the first being the informed consent. The statement that informed consent has been obtained has very little meaning unless the participant or his/her guardian is capable of understanding what is to be undertaken and unless all of hazards are made clear. If these are not known this, too, has been stated. Secondly, there is more reliable safeguard provided by presence of intelligent, informed, compassionate, conscientious, and responsible investigator.


REFERENCES:

1. Daniel Callahan and Bruce Jennings. Ethics and Public Health: Forging a Strong Relationship. American Journal of Public Health 2002; Vol 92, No. 2: 169-176.

2. Peter A Singer Et Al. Clinical Ethics Revisited. BMC Medical Ethics 2001; 2:1.

3. Steven S Coughlin. Ethical issues in epidemiologic research and public health practice. Emerging Themes in Epidemiology. BioMed central 2006; 3:16.

4. Nancy E. Kass. An Ethical Framework for Public Health. Public Health Matters.

5. Julie Myers Et Al. Privacy and Public Health at Risk: Public Health Confidentiality in the Digital Age. American Journal of Public Health 2008; Vol 98, No. 5:793-801.

6. Sandra Crouse Quinn. Protecting Human Subjects: the Role of Community Advisory Boards. American Journal of Public Health 2004; Vol 94, No. 6:918-922.

7. Henry K. Beecher. Ethics and Clinical Research. The New England Journal of Medicine 1966; Vol 274, No. 24:1354-1360.

8. C. Weijer and E.J. Emanuel. Protecting Communities in Biomedical Research. Science. Policy Forum: Ethics 2000; Vol 289:1142-1144.

Business ethics/lord of war quiz

The Lord of Communication: a VIDEO quiz

This is a first for my classes – a video quiz. I want you to create a video of yourself answering the quiz questions. Think of it like this: if we were in class, and I asked you to stand up and answer the first question, how would you respond? What would you say and how would you say it? How would you answer the question so we know that you have watched the film and you understand the business ethics concept? AND, just as importantly, how would you communicate your understanding to make OTHERS understand as well.

Below is the Lord of War quiz. There are directions on the quiz, so be sure you read them.

When answering the questions, please use as much detail and depth as you need to make your point clear to the audience. Your understanding of the characters, the film, and the business ethics concept should be explained using examples and detail from the film.

The bottom line is this: I want to be sure you have watched the film, and you understand all of the business ethics concepts we have gone over. The questions on the quiz will show me if you have watched the movie and whether you have gone through the lecture notes.

When you are ready, go to the “Student Videos/Speeches (Go React)” section to record your video quiz. There will be some directions there as well, so be sure to follow them.

GO REACT NEW_LOW VIDEO QUIZ_2020.pdf GO REACT NEW_LOW VIDEO QUIZ_2020.pdf – Alternative Formats

Critical Evaluation of Nova Scotias Provincial Prevention of Pressure Injuries Policy

Critical Evaluation of Nova Scotia’s Provincial Prevention of Pressure Injuries Policy

Table of Contents

Introduction…………………………………………………………..3

Historical context …………………………………………………….5

Pressure injuries……………………………………………………….6

Problem Definition……………………………………………………7

Indicators of the problem……………………………………………….8

Analysis of Goals and Objectives…………………………………….9

Analysis of Instruments and Implementation………………………………..10

Outcome Evaluation and Analysis…………………………………..10

Adequacy…………………………………………………….10

Suitability…………………………………………………….11

Accessibility…………………………………………….……12

Sustainability…………………………………………………12

Summary of analysis of criteria……………………………….12

Proposed method of Impact Evaluation……………………………….13

Summary and Future Recommendations…………………….……….14

INTRODUCTION

Healthy social policies are put in place to improve the health conditions of the people, to reduce morbidity and mortality rate such as government policy on prevention of pressure injuries and wound care treatment in Nova Scotia following a mortality issue in long term care. Pressure ulcer prevention protocol for long-term care settings was updated to challenge injustice that affected older adults in long term care (vulnerable group). The policy came into existence following the death of a lady in one of the long-term cares in Nova Scotia which occurred because of infected pressure injuries. This policy really interests me as a health professional in Nova Scotia.

The prevention and management of pressure injuries continues to be a concern in the Canadian long-term facilities. In 2003, Canadian Association of Wound Care (Wounds Canada) reported that the overall prevalence of pressure injuries across all long-term care settings was 26%, with approximately 70% of these pressure injuries was preventable by clinical practice, expert opinion, using best practices and use of appropriate equipment. Despite the focus on prevention to date, pressure injury incidence rates have not significantly reduced in Nova Scotia when compared with other provinces. A comprehensive approach focused on prevention is required across all areas of the health-care system to make a significant difference in incidence rates of pressure injuries. For the policy to be effective, interprofessional teams need to be integrated to include the person at risk of or with a pressure injury along with their families and departments such as purchasing and housekeeping. The cost of treating Pressure injuries is expensive. The lowest cost for treating a deep-tissue injury or Stage 1 or 2 wound is approximately $2,450 per month, while an uncomplicated Stage 3 or 4 is $3,616 per month Pressure injuries complicated by osteomyelitis cost approximately $12,648 per month to treat. The resources involved in the prevention of pressure injuries are less expensive than the cost of treatment.

The recommendations in the prevention of pressure injuries policy are based on the best available evidence and are intended to support the health care professional in planning and developing best practices in the prevention and management of pressure injuries.

Five priority action steps which are known as Wound prevention and management cycle were identified in the Policy; (1) Assess and Re assess- This serves as baseline information and helps in planning. Assess the resident, the wound, environmental and system challenges. Also, identify risk and causative factors that may affect skin integrity and wound healing. (2) Set Goals on prevention, healing, quality of life and symptom control. (3) Assemble the team, select membership based on resident need. (4) Establish and implement a plan of care that addresses the environment, system, the resident, the wound. Ensure effective communication among all members of the team. (5) Evaluate Outcomes. Ensure sustainability if goals are met and Re assess if goals are not met.

The policy’s objectives and directives are primarily focused on health care professionals and

clinicians to ensure that actions are carried out that will prevent pressure injuries throughout the province.

  • To examine the effectiveness of this policy, I will use an inclusion lens. An inclusion lens is used to determine if a policy is inclusive of all older adults, in particular those who belong to vulnerable populations, those who are disadvantaged, or who have been discriminated against by other policies or by society in general (Kwong., Lau, Lee, & Kwan, 2011). Saunders, Kraus, Peters, and Reed (2010) found out that the risk of developing pressure injuries increases with age. This is because aging comes with decreased skin elasticity, texture, circulation, cell replacement level and scarring process, and reduced peripheral sensitivity. Therefore, in evaluating this policy, I will look to see if older adults are included as effectively as those who are not disadvantaged or vulnerable and to determine if the policy has its intended benefits and effects on older adults.

The following criteria will be used to evaluate the prevention of pressure injuries policy; adequacy (i.e. are the measures being taken capable of solving the problem and of reaching the intended objectives and goals?); suitability (i.e. are the measures suitable for the older adults’ population; sustainability (i.e. can this policy effectively deal with the problem over a long period) and; accessibility (i.e. are the benefits of this policy accessible to all residents of long term facilities in the province of Nova Scotia?). Additionally, using these criteria I will conduct an outcome evaluation to determine if the policy is being effective in meeting its desired objectives (i.e. prevention of pressure injuries) in Nova Scotia.

Historical Context

Before delving further into the policy and beginning an evaluation, it is necessary to consider the history of prevention of pressure injuries in Nova Scotia.

In May 2016, the National Pressure Ulcer Advisory Panel updated the term pressure ulcer to pressure injury. The update was done to clarify that both Stage 1, stage 2 and Deep Pressure Injuries refer to intact skin while stage 3 and 4 are referred to as Open skin. Prevention of pressure injuries was part of the curriculum for teaching health care professionals in different level of studies. In 2012, Protocol document on prevention of pressure ulcers in long term care, Nova Scotia was developed by Department of Health and Wellness continuing care in collaboration with Nova Scotia Health Authority Provincial Wound Prevention and Management Program and Northwood as part of quality improvement initiative, the purpose of the policy is to enhance continuity of care and knowledge and build capacity of health workers to enhance pressure injury prevention and management in long term care facilities. The policy was updated in July 2018 following the death of a lady in one of the long-term cares in Nova Scotia which occurred because of infected pressure injuries.

Pressure Injuries

Woodbury (2014, p. 5) defined Pressure injuries (bed sores) as “injury to the skin and underlying tissue”. These injuries ranges from mild reddening of the skin to severe tissue damage and sometimes sepsis, cellulitis and infection that extends into muscle and bone. Government of Alberta (2018, P.1) described Pressure injuries in four stages:


  • Stage 1:

    there is skin discolouration, sores are not open wounds. The skin may be painful, but it has no breaks or tears. The skin appears reddened and does not blanch (lose colour briefly when you press your finger on it and then remove your finger). In a dark-skinned person, the area may appear to be a different colour than the surrounding skin, but it may not look red. Skin temperature is often warmer. And the stage 1 sore can feel either firmer or softer than the area around it.
  • At

    stage 2

    , the skin breaks open, wears away, or forms an ulcer, which is usually tender and painful. The sore expands into deeper layers of the skin. It can look like a scrape (abrasion), blister, or a shallow crater in the skin. Sometimes this stage looks like a blister filled with clear fluid. At this stage, some skin may be damaged beyond repair or may die.
  • During

    stage 3

    , the sore gets worse and extends into the tissue beneath the skin, forming a small crater. Fat may show in the sore, but not muscle, tendon, or bone.
  • At

    stage 4

    , the pressure injury is very deep, reaching into muscle and bone and causing extensive damage. Damage to deeper tissues, tendons, and joints may occur.

Braden scale is an important component in prevention of pressure injuries as it helps in predicting pressure injuries risk among older adults. The Braden scale uses six criteria which includes: Moisture, activity, friction shear, sensory perception, moisture and mobility. Each criterion is rated on a scale of 1 to 4, except the ‘friction and shear’ criteria which is rated on a 1-3 scale which makes the possible total of 23 points. Higher score means a lower risk of developing a pressure ulcer and lower score means a higher risk of developing pressure ulcer (NSHA, 2018). The Braden Scale assessment score scale:

  • Very High Risk: Total Score 9 or less
  • High Risk: Total Score 10-12
  • Moderate Risk: Total Score 13-14
  • Mild Risk: Total Score 15-18
  • No Risk: Total Score 19-23

Problem Definition

Saunders et al. (2010) found out that the risk of developing pressure injuries increases with age. This is because aging comes with decreased skin elasticity, texture, circulation, cell replacement level and scarring process, and reduced peripheral sensitivity. Also, Lannering, Ernsth, and Johansson (2017) reported that poor nutritional status is a major contributor to development of pressure injuries in older adults. If someone is not taking the required amount of nutrients the body needs, it makes the body to be more vulnerable to wear and tear. In the same vein, Long-term care residents may not be able to take enough food due underling medical conditions like difficulty with swallowing, lack of appetite, or because they are living with dementia which has made them to lose interest in eating or the knowledge of how to feed themselves. In addition, Woodbury (2014) identified Diabetes as one of the risk factors for developing pressure injuries. For instance, many residents with diabetes have poor circulation to the feet. If such resident develops an ulcer on the foot, it may not heal due to lack of oxygen. Furthermore, long term facilities staffing policy which has not increased since 1988 has made it stressful for staffs of long-term facilities to effectively care for residents in order to prevent pressure injuries such as engaging in regular turning of residents, assessment of wounds.

Indicators of the problem

To evaluate this policy, we can use different ways which may include:

how well the policy has been implemented and if its implementation is appropriate for the policy

goals and tools utilized; whether long-term care facilities are actually delivering the policy

consistently and appropriately along with analysis of the policy’s components (process

evaluation), a cost-benefit analysis or whether the policy is achieving its goals and

making a difference (outcome evaluation).

Unfortunately, Nova Scotia Health Authority has not published recent data on the province pressure injuries. The data I used in this paper was gotten from one of the NSHA new employee orientation slides as I have the privilege of getting hired recently. The following indicators will be used to evaluate the goals of prevention of pressure injuries policy in Nova Scotia; Incidence (percentage of residents with a new pressure injury in a 6-month period)- In April to October 2018, Long term facilities reported a total of 291 bedsores — with 52 at the severe stage 3 and 4 levels. Prevalence (percentage of residents with a pressure injury in a 6-month period)- In October 2018, the prevalence rate for pressure injuries averaged seven per cent for the province. Percentage of residents with a healed pressure injury in a 12-week period, percentage of residents with a healed pressure injury who were diagnosed with a secondary pressure injury within 1 year (recurrence), percentage of residents with a pressure injury who had a diagnosed wound infection in a 6-month period , and percentage of residents with a pressure injury in a 12-month period who reported high satisfaction with the care provided.

Analysis of the Goals and Objectives

The overall goal of the prevention of pressure injuries policy in Nova Scotia is quality improvement initiative. Its objective is to enhance continuity of care and knowledge and build capacity of health workers to enhance pressure injury prevention and management in long term care facilities. This is to reduce morbidity and mortality rate among residents of long-term care facilities ( Woodbury & Houghton, 2014).

When considering the definition of the problem, high incidence of pressure injuries in Nova Scotia, part of the contributing factor is the staffing policy that have not increased since 1988 when the long-term facilities had fewer residents and now when they have more residents to take care.

Additionally, the best way to achieve this is by increasing the long-term staff strength, retraining of health care workers on wound care management and prevention of pressure ulcers, turning bedridden residents every hour, application of barrier ointment to prominent bony prominences’, adequate hydration of residents, and improved documentation and reporting.

However, the policy is not a specific to a group of people but, rather a general approach and knowledge to all race, ethnicity, religion, sex, gender, age, mental and physical disability.

Analysis of Instruments and Implementation

Department of Health and Wellness continuing care in collaboration with Nova Scotia Health Authority Provincial Wound Prevention and Management Program and Northwood developed a protocol document that involves steps and directives to be followed by health care professionals that will purportedly lead to decreased incidence of pressure injuries and prevention of pressure injuries in Nova Scotia. Thus, the primary instrument utilized by the policy is regulation.

These regulations which is more of education do not apply to the residents and their families but rather to health care professionals. These regulations include retraining of health care workers on wound care management and prevention of pressure ulcers, turning bedridden residents every hour, application of barrier ointment to prominent bony prominences’, adequate hydration of residents, improved documentation and reporting.

Outcome Evaluation and Analysis

Earlier it was mentioned that to evaluate this policy, an outcome evaluation would be conducted

considering four criteria: adequacy, suitability, sustainability and accessibility. These have been

touched on in previous sections, but it is now appropriate to look at each in more detail.

Adequacy

Is this policy adequate; are the instruments employed capable of effecting the change that

we are looking for? In theory, all bases are covered by regulation, public information and

government/non-government partnerships and accompanying programs. Residents and families are made aware of the benefits of prevention of pressure injuries. Long term care facilities are required to share this information with Residents and their families. As earlier mentioned, the policy consists of five priority action steps which are known as Wound prevention and management cycle. Health care professionals’ partner with Resident and families to achieve optimum results. Proper documentation and reporting of new cases of pressure injuries will help in determining if the policy is adequate or not.

Suitability

Are the programs within this policy suitable for all residents of long term facilities (race, ethnicity, religion, age, sex, gender, mental and physical disability) across the province? To truly

answer this question, it would be necessary to conduct a focus group interviews among health care professionals, Residents and families to determine the suitability of the policy. Studies found out that people with low economic status are at higher risk of developing pressure injuries compared to people with high economic status. Possible explanations for this, is that high income class can afford some resources that might help to prevent pressure injuries such as customized wheel chair and heel protectors and take good balanced diet that help in prevention of pressure ulcers. I think since prevention of pressure injuries is education driven, it provided an equal access for people from both low and high economic status. With or without availability of customized wheelchair or heel protectors, irrespective of socio-economic status, all residents that are bed ridden need to be assessed, turned every hour, application of barrier creams and heel should be elevated using pillows.

Accessibility

Are the programs implemented in this policy accessible to all residents of long-term facilities in the province? I think since prevention of pressure injuries is more of education for health care professionals, the protocol on prevention of pressure injuries would be accessible to all residents of long-term facilities in Nova Scotia. Everyone is to be treated the same way in order to prevent pressure injuries.

Sustainability

Finally, is the instruments, and policy programs sustainable? In addition, is our health care system in Canada sustainable? given that many of the programs that are part of this policy are free and are offered through the Department of Health and wellness for the province. However, that is too much to get into for the purpose of this paper. Of the three instruments utilized by this policy, the most sustainable are the directives. Partnerships with professional bodies such as college of Registered Nurses, College of Licensed Practical Nurses, College of Physicians to incorporate prevention of pressure injuries as part of requirements for annual license renewal will enhance sustainability of the policy. Also, engaging the media to sensitize the general public on prevention of pressure injuries will help to sustain the policy

Summary of criteria analysis

In conclusion, I feel that this policy is a positive step towards prevention of pressure injuries

in the province of Nova Scotia, but having a policy alone is not enough. All the

instruments employed, from the directives, to the public awareness, to the partnerships with professional bodies and programs must be continually assessed to ensure that they are meeting the

changing needs of Nova Scotia population.

Proposed Method of Outcome Evaluation

To conduct an outcome evaluation, using these criteria I will conduct an outcome evaluation to determine if the policy is being effective in meeting its desired objectives (i.e. prevention of pressure injuries) in Nova Scotia.

we need to know if the policy and the policy have had their intended effects. That is, is the policy successfully achieving its desired objectives and goals (i.e. prevention of pressure injuries in Nova Scotia?). In this case the overall aim is prevention of pressure injuries in long term care facilities in Nova Scotia and, a decrease in overall incidence and prevalence of pressure injuries.

The following indicators will be used to evaluate the goals of prevention of pressure injuries policy in Nova Scotia; Incidence (percentage of residents with a new pressure injury in a 6-month period) , Prevalence (percentage of residents with a pressure injury in a 6-month period), percentage of residents with a healed pressure injury in a 12-week period, percentage of residents with a healed pressure injury who were diagnosed with a secondary pressure injury within 1 year (recurrence), percentage of residents with a pressure injury who had a diagnosed wound infection in a 6-month period , and percentage of residents with a pressure injury in a 12-month period who reported high satisfaction with the care provided. Proper reporting and documentation by health care professionals of long-term care facilities will enhance the success of these indicators.

Summary and Future Recommendations

In the last six months, incidence of pressure injuries in long term care facilities in Nova Scotia have greatly reduced by 4.3 percent (Chronicle Herald news: 04.09.18). For some reasons, although statistics regarding the number of pressure injuries in Nova Scotia are not officially published by the Government so little information is known about the number of individuals in Nova scotia who have pressure injuries. Such information is important to assess the scope and healthcare costs of pressure injuries and develop public policies (Woodbury, 2014). To obtain estimated pressure injuries prevalence rates in Nova Scotia, existing data (gathered between 2008 and 2018) from different long-term care settings across the province were obtained from peer-reviewed published studies and from unpublished studies provided by individuals and pressure injuries support.

Policy’s primary instruments and their implementation have been very well executed overall across the province, there are still some areas that needs improvement. Most especially, the area of reporting and documentation as this is important to determine the success of the policy.

REFERENCES

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Select a theory or conceptual model relevant to your research interest.

Select a theory or conceptual model relevant to your research interest.

  1. Select a theory or conceptual model relevant to your research interest. Determine how the key concepts or variables of interest relate to each other within the selected framework.
  2. If the author explains their theory through a diagram, explain what each part of the diagram means.
  3. Write a paper explaining the model – generally and specifically applied to your topic of interest. Examine strengths and weaknesses of the selected model in general, and as it applies to your research topic. Hint: think about how you would explain this if you were teaching a novice about the theory.